This is relative to previous findings of prolactin being a stimulatory agent of IgA synthesis [27]
This is relative to previous findings of prolactin being a stimulatory agent of IgA synthesis [27]. Elevated galactosylation from the
This is relative to previous findings of prolactin being a stimulatory agent of IgA synthesis [27]. Elevated galactosylation from the
Therefore, we presumed that anti-SSA antibody-associated autoimmunity may have exacerbated the hypofunction of NCCTs, resulting in the introduction of GS.
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